antibodies against ki67 (Servicebio Inc)
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Antibodies Against Ki67, supplied by Servicebio Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/antibodies+against+ki67/anti+ki67+%E2%80%A2/pm42085840-54-0-12
Average 86 stars, based on 1 article reviews
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Activity Assay:Article Title: M1-like tumor-associated macrophages promote colorectal cancer progression through feedback activation of the IL1β/NF-κB signaling activity. Article Snippet: Tumor-associated macrophages (TAMs) are the main tumor infiltrating immune cell types in tumor microenvironment (TME) that have related to tumor progression.. However, crosstalk between TAMs and colorectal cancer (CRC) cells remains poorly understood.. In this study, conditioned media (CM) was harvested from CRC cells or activated TAMs. Blocking Assay:Article Title: M1-like tumor-associated macrophages promote colorectal cancer progression through feedback activation of the IL1β/NF-κB signaling activity. Article Snippet: Tumor-associated macrophages (TAMs) are the main tumor infiltrating immune cell types in tumor microenvironment (TME) that have related to tumor progression.. However, crosstalk between TAMs and colorectal cancer (CRC) cells remains poorly understood.. In this study, conditioned media (CM) was harvested from CRC cells or activated TAMs. Incubation:Article Title: M1-like tumor-associated macrophages promote colorectal cancer progression through feedback activation of the IL1β/NF-κB signaling activity. Article Snippet: Tumor-associated macrophages (TAMs) are the main tumor infiltrating immune cell types in tumor microenvironment (TME) that have related to tumor progression.. However, crosstalk between TAMs and colorectal cancer (CRC) cells remains poorly understood.. In this study, conditioned media (CM) was harvested from CRC cells or activated TAMs. Article Title: A Versatile Approach to Synergistically Combine Physical Barriers and Bioactive Agents for Intrauterine Adhesion Prevention Article Snippet: .. The tissue sections were incubated overnight at 4 °C with primary Article Title: Employing epigenetic protein degradation techniques to block CCL5-mediated photodynamic therapy via a programmed delivery platform. Article Snippet: .. For IHC, the slides were subsequently separately stained with primary Article Title: Employing epigenetic protein degradation techniques to block CCL5-mediated photodynamic therapy via a programmed delivery platform Article Snippet: .. For IHC, the slides were subsequently separately stained with primary Article Title: DNASE1L3 regulates macrophage polarization through the NLRP3-GSDMD signaling pathway affecting hepatocellular carcinoma progression Article Snippet: Received: Jun 2, 2025 Revised: Oct 27, 2025 Accepted: Nov 17, 2025 ABSTRACT Hepatocellular carcinoma (HCC) remains a lethal malignancy with a persistently poor prognosis.. While our previous studies established the anti-tumor function of Deoxyribonuclease I Like Protein 3 (DNASE1L3) in HCC, the underlying mechanisms involving the immune microenvironment are less understood.. Here, we demonstrate that loss of DNASE1L3 accelerated HCC progression by impairing M1-type macrophage polarization in Dnase1l3 knockout (KO) mice, co-culture models, RNA-seq, and comprehensive molecular/cellular analyses. Reverse Transcription Polymerase Chain Reaction:Article Title: Cauloside A interacts with TLR4 to induce JNK/Caspase-3/GSDME-dependent pyroptosis in non-small cell lung cancer cells. Article Snippet: With non-small cell lung cancer (NSCLC) accounts for the majority of diagnoses, lung cancer continues to be the most common and deadly type of cancer worldwide.. While treatment options have advanced, therapeutic efficacy remains constrained by drug resistance and side effects, highlighting the demand for novel therapeutic agents.. Cauloside A, a natural compound isolated from Fructus Akebiae, has shown anti-tumor potential; however, its precise mechanism of action in NSCLC has remained unclear. Immunohistochemistry:Article Title: Employing epigenetic protein degradation techniques to block CCL5-mediated photodynamic therapy via a programmed delivery platform. Article Snippet: .. For IHC, the slides were subsequently separately stained with primary Article Title: Employing epigenetic protein degradation techniques to block CCL5-mediated photodynamic therapy via a programmed delivery platform Article Snippet: .. For IHC, the slides were subsequently separately stained with primary Staining:Article Title: Employing epigenetic protein degradation techniques to block CCL5-mediated photodynamic therapy via a programmed delivery platform. Article Snippet: .. For IHC, the slides were subsequently separately stained with primary Article Title: Employing epigenetic protein degradation techniques to block CCL5-mediated photodynamic therapy via a programmed delivery platform Article Snippet: .. For IHC, the slides were subsequently separately stained with primary Immunohistochemical staining:Article Title: DNASE1L3 regulates macrophage polarization through the NLRP3-GSDMD signaling pathway affecting hepatocellular carcinoma progression Article Snippet: Received: Jun 2, 2025 Revised: Oct 27, 2025 Accepted: Nov 17, 2025 ABSTRACT Hepatocellular carcinoma (HCC) remains a lethal malignancy with a persistently poor prognosis.. While our previous studies established the anti-tumor function of Deoxyribonuclease I Like Protein 3 (DNASE1L3) in HCC, the underlying mechanisms involving the immune microenvironment are less understood.. Here, we demonstrate that loss of DNASE1L3 accelerated HCC progression by impairing M1-type macrophage polarization in Dnase1l3 knockout (KO) mice, co-culture models, RNA-seq, and comprehensive molecular/cellular analyses. Labeling:Article Title: DNASE1L3 regulates macrophage polarization through the NLRP3-GSDMD signaling pathway affecting hepatocellular carcinoma progression Article Snippet: Received: Jun 2, 2025 Revised: Oct 27, 2025 Accepted: Nov 17, 2025 ABSTRACT Hepatocellular carcinoma (HCC) remains a lethal malignancy with a persistently poor prognosis.. While our previous studies established the anti-tumor function of Deoxyribonuclease I Like Protein 3 (DNASE1L3) in HCC, the underlying mechanisms involving the immune microenvironment are less understood.. Here, we demonstrate that loss of DNASE1L3 accelerated HCC progression by impairing M1-type macrophage polarization in Dnase1l3 knockout (KO) mice, co-culture models, RNA-seq, and comprehensive molecular/cellular analyses. Light Microscopy:Article Title: DNASE1L3 regulates macrophage polarization through the NLRP3-GSDMD signaling pathway affecting hepatocellular carcinoma progression Article Snippet: Received: Jun 2, 2025 Revised: Oct 27, 2025 Accepted: Nov 17, 2025 ABSTRACT Hepatocellular carcinoma (HCC) remains a lethal malignancy with a persistently poor prognosis.. While our previous studies established the anti-tumor function of Deoxyribonuclease I Like Protein 3 (DNASE1L3) in HCC, the underlying mechanisms involving the immune microenvironment are less understood.. Here, we demonstrate that loss of DNASE1L3 accelerated HCC progression by impairing M1-type macrophage polarization in Dnase1l3 knockout (KO) mice, co-culture models, RNA-seq, and comprehensive molecular/cellular analyses. |
